Loss of Aatf in renal tubular cells results in cystic kidney disease
We investigate the pathogenesis of polycystic kidney disease (PKD) and other ciliopathies. Our focus lies in the study of pkd and cilia proteins in various systems to understand what changes in signal transduction explain the disease phenotypes.
Associations and Support:
- KFO 201
- Else-Kröner-Fresenius Stiftung
- European Renal Association (ERA)
- European Molecular Biology Organization (EMBO)